Arrested preoligodendrocyte maturation contributes to myelination failure in premature infants
Identifieur interne : 001466 ( Main/Exploration ); précédent : 001465; suivant : 001467Arrested preoligodendrocyte maturation contributes to myelination failure in premature infants
Auteurs : Joshua R. Buser [États-Unis] ; Jennifer Maire [États-Unis] ; Art Riddle [États-Unis] ; Xi Gong [États-Unis] ; Thuan Nguyen [États-Unis] ; Kerst Nelson [États-Unis] ; Ning Ling Luo [États-Unis] ; Jennifer Ren [États-Unis] ; Jaime Struve [États-Unis] ; Larry S. Sherman [États-Unis] ; Steven P. Miller [Canada] ; Vann Chau [Canada] ; Glenda Hendson [Canada] ; Praveen Ballabh [États-Unis] ; Marjorie R. Grafe [États-Unis] ; Stephen A. Back [États-Unis]Source :
- Annals of Neurology [ 0364-5134 ] ; 2012-01.
Abstract
The major form of magnetic resonance imaging–defined white matter injury (WMI) comprises diffuse lesions where the burden of small necrotic foci (microscopic necrosis) is poorly defined. We hypothesized that myelination failure associated with diffuse WMI involves an aberrant injury response linked to arrested preoligodendrocyte (preOL) maturation in reactive astrocyte‐rich lesions.
Url:
DOI: 10.1002/ana.22627
Affiliations:
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<front><div type="abstract">The major form of magnetic resonance imaging–defined white matter injury (WMI) comprises diffuse lesions where the burden of small necrotic foci (microscopic necrosis) is poorly defined. We hypothesized that myelination failure associated with diffuse WMI involves an aberrant injury response linked to arrested preoligodendrocyte (preOL) maturation in reactive astrocyte‐rich lesions.</div>
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